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Abstract
Background
Patients with congenital long QT syndrome (LQTS) are at increased risk of ventricular arrhythmia, particularly during labour and the puerperium.
Case
A 28-year-old primigravida with known LQTS underwent induction of labour at 41 weeks’ gestation using a Foley catheter balloon and IV oxytocin. Vaginal delivery with passive second stage and outlet forceps was undertaken with early epidural analgesia to prevent tachycardia and psychological stress. The patient gave birth to a healthy female, and had an uncomplicated postpartum period under continuous electrocardiogram monitoring.
Conclusion
Vaginal delivery with use of oxytocin for the induction of labour can be safely undertaken in patients with LQTS.
Résumé
Contexte
Les patientes qui présentent le syndrome du QT long congénital (syndrome du QTL) sont exposées à un risque accru d’arythmie ventriculaire, particulièrement pendant le travail et la puerpéralité.
Cas
Une primigravide de 28 ans présentant un syndrome du QTL connu a subi un déclenchement du travail à 41 semaines de gestation effectué au moyen d’une sonde de Foley à ballonnet et d’une perfusion d’oxytocine. Un accouchement vaginal (dans le cadre duquel le 2e stade a été passif et où l’on a procédé à une extraction par application de forceps à la partie basse) a été mis en oeuvre et une analgésie péridurale a été pratiquée de façon précoce pour prévenir la tachycardie et le stress psychologique. La patiente a donné naissance à une fille en santé; elle a par la suite connu une période postpartum exempte de complications sous surveillance électrocardiographique continue.
Conclusions
Un accouchement vaginal s’accompagnant d’un déclenchement du travail au moyen d’oxytocine peut être mis en oeuvre en toute sécurité chez les patientes qui présentent un syndrome du QTL.
Key Words
References
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Article info
Publication history
Accepted:
July 18,
2012
Received:
July 9,
2012
Footnotes
Competing Interests: None declared.
Identification
Copyright
© 2012 Society of Obstetricians and Gynaecologists of Canada. Published by Elsevier Inc. All rights reserved.
